Aging begins at the cellular level, primarily around the age of 25, when cell turnover slows down.
While the exact mechanisms of aging are complex and still being researched, several factors contribute to this initial slowdown, leading to observable effects over time:
-
Slowing Cell Turnover: Cells are constantly being replaced. As we age, the rate at which old or damaged cells are replaced with new ones decreases. This reduced turnover leads to a gradual decline in tissue and organ function.
-
Decreased Production of Key Proteins: The production of vital structural proteins like collagen, elastin, and hyaluronic acid diminishes. These substances are crucial for maintaining skin elasticity, firmness, and hydration. Their decline contributes to wrinkles, sagging, and dryness.
-
Mitochondrial Dysfunction: Mitochondria, the powerhouses of cells, become less efficient with age. This reduced energy production affects cellular functions and contributes to the aging process.
-
DNA Damage Accumulation: Over time, DNA accumulates damage from various sources (e.g., UV radiation, environmental toxins). This damage can impair cellular function and contribute to aging.
-
Stem Cell Exhaustion: Stem cells play a critical role in tissue repair and regeneration. As we age, the number and function of stem cells decline, limiting the body's ability to repair itself.
In summary, aging commences at the cellular level with a slowing of cell turnover and a decline in the production of essential substances like collagen, elastin, and hyaluronic acid, compounded by mitochondrial dysfunction, DNA damage, and stem cell exhaustion. These changes collectively contribute to the visible and functional signs of aging.
