Psoriasis is rooted in a malfunctioning immune system.
Understanding the Immune System's Role in Psoriasis
The rapid skin cell turnover characteristic of psoriasis isn't fully understood, but current research strongly points to an immune system problem as the underlying cause. Normally, skin cells regenerate slowly. However, in individuals with psoriasis, the immune system triggers an accelerated cell growth cycle. This leads to the characteristic buildup of thick, scaly skin patches. While the exact trigger remains unknown, the immune system's misfiring is the core issue.
Key Aspects of the Immune System Dysfunction:
- Accelerated Cell Growth: The immune system mistakenly signals for the rapid production of new skin cells. This process happens much faster than in healthy skin.
- Inflammation: This accelerated cell growth is accompanied by significant inflammation, contributing to the redness, itching, and pain associated with psoriasis.
- Genetic Predisposition: While the precise mechanism isn't fully elucidated, a genetic component is strongly implicated, increasing susceptibility to this immune system dysfunction.
The body's natural process of creating new skin cells in the deepest layer is drastically sped up, resulting in the visible symptoms of psoriasis. More research is needed to pinpoint the precise immune system triggers and pathways involved.
