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Why Does Stress Cause Mouth Ulcers?

Published in Stress and Oral Health 2 mins read

Stress can contribute to the development of mouth ulcers, also known as canker sores, through a few key mechanisms.

How Stress Leads to Mouth Ulcers

The primary connection between stress and mouth ulcers involves physical and biological responses to stress.

Physical Injury

  • Cheek and Lip Biting: When stressed, people might subconsciously engage in habits like biting their cheeks or lips. This physical trauma can injure the delicate oral mucosa and initiate the formation of an ulcer, as mentioned in the reference.

    "Because of the stressful condition, patients may injure their oral mucosa by biting their cheeks or lips, which leads to the development of oral ulcers."

Biological Pathways

  • Genetic Alterations: The body's reaction to stress can also alter genetic pathways, potentially making individuals more susceptible to oral ulcers. The reference highlights that changes in these pathways can be linked to stressful reactions.

    "Alterations in the genetic pathways may also be linked to stressful reactions that occur within the body."

Reduced Immunity

  • Compromised Immune System: Chronic stress can weaken the immune system, making it harder for the body to fight off infections and heal oral sores. Though not directly mentioned in the provided reference, it is a well-established factor in ulcer development.

Management and Prevention

While avoiding stress completely might be impractical, strategies can help manage stress and reduce the occurrence of mouth ulcers:

  • Stress Management Techniques:
    • Regular exercise
    • Mindfulness and meditation
    • Deep breathing exercises
    • Adequate sleep
  • Avoid Oral Trauma:
    • Be mindful of habits like cheek or lip biting
    • Use a soft-bristled toothbrush
  • Maintain Oral Hygiene:
    • Brush and floss regularly
    • Use a mouthwash recommended by your dentist

In summary, stress contributes to mouth ulcers through physical injury (biting) and potentially by altering genetic pathways associated with the body's stress responses.

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